Case study on Eisenmenger Syndrome

Abstract

The most severe type of pulmonary arterial hypertension, known as Eisenmenger syndrome, is caused by congenital heart disease with systemic-to-pulmonary shunt. Patients with Eisenmenger syndrome experience a complex and multisystemic disorder that includes coagulation disorders (bleeding complications and paradoxical embolisms), renal dysfunction, hypertrophic osteoarthropathy, heart failure, reduced quality of life, and premature death due to the chronic slow progressive hypoxemia with central cyanosis. The present case of 31-year-old female patient who was admitted in the cardiology department of a tertiary care teaching hospital with complaints of shortness of breath, increased excretion, cough with mucoid expectoration and discolouration of eye, tongue and fingers. Treatment includes antibacterial, diuretics, mucolytic agents, phosphodiesterase (PDE) inhibitors. After the continuous treatment for a period of 7 days the patient was found to be normal with no major complaints and so, the pharmacist when performed discharge counselling and patient was discharged with a 2week drug regimen after discharge.